Search results for "genetics [Genetic Variation]"

showing 10 items of 3029 documents

Idiopathic Non-Dental Facial Pain Syndromes in Italian Children: A Clinical Case Series

2023

Background. The orofacial pain syndromes (OFPs) are a heterogeneous group of syndromes characterized by painful attacks involving the orofacial structures. They may be summarily subdivided into two great categories: (1) orofacial pain mainly attributed to dental disorders such as dentoalveolar and myofascial orofacial pain or temporomandibular joint (TM) pain; (2) orofacial pain mainly attributed to non-dental pain as neuralgias, facial localization of primary headaches or idiopathic orofacial pain. The second group is uncommon, often described by single case reports, can often show overlapping symptoms with the first group, and represents a clinical challenge, carrying the risk of underval…

childrenSpace and Planetary Scienceorofacial pain; children; headache; red ear syndrome; migraine; trigeminal autonomic syndromesorofacial painred ear syndrometrigeminal autonomic syndromesPaleontologymigraineheadacheGeneral Biochemistry Genetics and Molecular BiologyEcology Evolution Behavior and SystematicsLife
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Removal of Chromophore-proximal Polar Atoms Decreases Water Content and Increases Fluorescence in a Near Infrared Phytofluor

2015

Genetically encoded fluorescent markers have revolutionized cell and molecular biology due to their biological compatibility, controllable spatiotemporal expression, and photostability. To achieve in vivo imaging in whole animals, longer excitation wavelength probes are needed due to the superior ability of near infrared light to penetrate tissues unimpeded by absorbance from biomolecules or autofluorescence of water. Derived from near infrared-absorbing bacteriophytochromes, phytofluors are engineered to fluoresce in this region of the electromagnetic spectrum, although high quantum yield remains an elusive goal. An invariant aspartate residue is of utmost importance for photoconversion in…

chromophore binding domain (CBD)Analytical chemistryQuantum yieldPhotochemistryBiochemistry Genetics and Molecular Biology (miscellaneous)BiochemistryFluorescence spectroscopychemistry.chemical_compoundDeinococcus radioduransWiPhy2Side chainMolecular Biologylcsh:QH301-705.5Wisconsin infrared phytofluor (WiPhy2)Original ResearchBiliverdinta114Physicsta1182Excitation-emission matrix (EEM)ChromophorePhotobleachingFluorescenceexcitation-emission matrix (EEM)chemistrylcsh:Biology (General)Excited statetetrapyrroleFrontiers in Molecular Biosciences
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Hair Cortisol Concentration as a Biomarker of Sleep Quality and Related Disorders

2021

Cortisol is the end product of the hypothalamic-pituitary-adrenal (HPA) axis, and its production is increased mainly in stressful situations or in chronic disorders accompanied by stress enhancement. Altered cortisol concentrations have been reported in a number of neuropsychiatric diseases and sleep disorders. Cortisol concentrations have been measured using several methods, and in several matrixes, such as blood, saliva, and urine. However, lately, hair cortisol, for several reasons, has emerged as a promising biomarker of long-term retrospective HPA activation. Several experimental approaches for cortisol measurement with the corresponding concentration reference ranges and a summary of …

circadian rhythmSalivaendocrine systemUrinary systeminsomniaPhysiologyUrineReviewGeneral Biochemistry Genetics and Molecular BiologyShift work03 medical and health sciences0302 clinical medicinechildrenInsomniaMedicineCircadian rhythmlcsh:ScienceEcology Evolution Behavior and Systematicsbusiness.industryPaleontologySleep in non-human animals030227 psychiatryshift workSpace and Planetary ScienceBiomarker (medicine)lcsh:QREM sleepmedicine.symptombusiness030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsLife
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Prevention of Dominant IgG Adsorption on Nanocarriers in IgG‐Enriched Blood Plasma by Clusterin Precoating

2019

Abstract Nanocarriers for medical applications must work reliably within organisms, independent of the individual differences in the blood proteome. Variation in the blood proteome, such as immunoglobulin levels, is a result of environmental, nutrition, and constitution conditions. This variation, however, should not influence the behavior of nanocarriers in biological media. The composition of the protein corona is investigated to understand the influence varying immunoglobulin levels in the blood plasma have on the interactions with nanocarriers. Specifically, the composition of the nanocarriers' coronas is analyzed after incubation in plasma with normal or elevated immunoglobulin G (IgG)…

clusterinGeneral Chemical Engineeringmedia_common.quotation_subjectGeneral Physics and AstronomyMedicine (miscellaneous)Protein Corona02 engineering and technology010402 general chemistry01 natural sciencesBiochemistry Genetics and Molecular Biology (miscellaneous)Immunoglobulin Gimmunoglobulin Gprotein coronaBlood plasmaGeneral Materials ScienceReceptorInternalizationlcsh:Sciencemedia_commonstealth effectbiologyFull PapernanocarriersChemistryGeneral EngineeringFull Papers021001 nanoscience & nanotechnology0104 chemical sciencesIgG bindingBiophysicsbiology.proteinlcsh:QNanocarriersAntibody0210 nano-technologyAdvanced Science
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Zebrafish Fins as a Model System for Skeletal Human Studies

2007

Recent studies on the morphogenesis of the fins ofDanio rerio(zebrafish) during development and regeneration suggest that a number of inductive signals involved in the process are similar to some of those that affect bone and cartilage differentiation in mammals and humans. Akimenko et al. (2002) has shown that bone morphogenetic protein-2b (BMP2b) is involved in the induction of dermal bone differentiation during fin regeneration. Many other groups have also shown that molecules from the transforming growth factor-beta superfamily (TGFβ), including BMP2, are effective in promoting chondrogenesis and osteogenesisin vivoin higher vertebrates, including humans. In the present study, we review…

collagenPathologylcsh:MedicineReview Articlebonelcsh:TechnologydentineExtracellular matrixbone regenerationOsteogenesisMorphogenesislcsh:ScienceZebrafishZebrafishGeneral Environmental Sciencetransforming growth factor betaDermal bonebiologyenamelGeneral MedicineCell biologyendochondral ossificationmedicine.anatomical_structureModels Animalmedicine.medical_specialtyextracellular matrixosteocyteregenerative medicineray dermal boneBone morphogenetic protein 2Bone and BonesGeneral Biochemistry Genetics and Molecular BiologyFin regenerationsonic hedgehogbone morphogenetic proteinsmedicineAnimalsHumansactinopterygian fishesmammalslepidotrichiascleroblastmesenchymal stem cellslcsh:TRegeneration (biology)Cartilagelcsh:RZebrafish Proteinsbone repairbiology.organism_classificationChondrogenesisCartilageregenerationintramembranous ossificationlcsh:Qcell therapyvertebratesThe Scientific World Journal
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Genetic and Chemical Modifiers Of A CUG Toxicity Model in Drosophila

2007

Non-coding CUG repeat expansions interfere with the activity of human Muscleblind-like (MBNL) proteins contributing to myotonic dystrophy 1 (DM1). To understand this toxic RNA gain-of-function mechanism we developed a Drosophila model expressing 60 pure and 480 interrupted CUG repeats in the context of a non-translatable RNA. These flies reproduced aspects of the DM1 pathology, most notably nuclear accumulation of CUG transcripts, muscle degeneration, splicing misregulation, and diminished Muscleblind function in vivo. Reduced Muscleblind activity was evident from the sensitivity of CUG-induced phenotypes to a decrease in muscleblind genetic dosage and rescue by MBNL1 expression, and furthe…

congenital hereditary and neonatal diseases and abnormalitiesGene Dosagelcsh:MedicineRNA-binding proteinBiologyEyechemistry.chemical_compoundTrinucleotide RepeatsAnimalsDrosophila ProteinsMyotonic DystrophyMBNL1lcsh:ScienceGeneGenetics and Genomics/Genetics of DiseaseGeneticsMessenger RNADNA Repeat ExpansionMultidisciplinaryAlternative splicinglcsh:RBrainNuclear ProteinsRNA-Binding ProteinsRNAPhenotypeCell biologyDisease Models AnimalGenetics and Genomics/Disease ModelschemistryRNA splicingDrosophilalcsh:QGenèticaResearch Article
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Huntingtin controls neurotrophic support and survival of neurons by enhancing BDNF vesicular transport along microtubules.

2004

AbstractPolyglutamine expansion (polyQ) in the protein huntingtin is pathogenic and responsible for the neuronal toxicity associated with Huntington's disease (HD). Although wild-type huntingtin possesses antiapoptotic properties, the relationship between the neuroprotective functions of huntingtin and pathogenesis of HD remains unclear. Here, we show that huntingtin specifically enhances vesicular transport of brain-derived neurotrophic factor (BDNF) along microtubules. Huntingtin-mediated transport involves huntingtin-associated protein-1 (HAP1) and the p150Glued subunit of dynactin, an essential component of molecular motors. BDNF transport is attenuated both in the disease context and b…

congenital hereditary and neonatal diseases and abnormalitiesHuntingtinCell SurvivalContext (language use)Nerve Tissue ProteinsMicrotubulesModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyMiceNeurotrophic factorsmental disordersHuntingtin ProteinAnimalsCells CulturedNeuronsHuntingtin ProteinbiologyBiochemistry Genetics and Molecular Biology(all)Huntingtin-associated protein 1Brain-Derived Neurotrophic FactorCytoplasmic VesiclesBrainNuclear ProteinsBiological TransportDynactin ComplexCell biologynervous system diseasesVesicular transport proteinDNA-Binding ProteinsBiochemistrynervous systembiology.proteinDynactinMicrotubule-Associated ProteinsNeurotrophinCell
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Outcome of patients with classical infantile pompe disease receiving enzyme replacement therapy in Germany

2015

Enzyme replacement therapy (ERT) has been shown to improve outcome in classical infantile Pompe disease. The purpose of this study was to assess mortality, morbidity, and shortcomings of ERT in a larger cohort of patients treated outside clinical trials. To accomplish this, we retrospectively analyzed the data of all 23 subjects with classical infantile Pompe disease having started ERT in Germany between January 2003 and December 2010.Ten patients (43%) deceased and four others (17%) became ventilator dependent. Seven infants (30.5%) made no motor progress at all, while seven (30.5%) achieved free sitting, and nine (39%) gained free walking. Besides all the seven patients (100%) attaining n…

congenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtybusiness.industryMEDLINEnutritional and metabolic diseases610 Medicine & healthDiseaseMetabolic myopathyEnzyme replacement therapymedicine.disease1301 Biochemistry Genetics and Molecular Biology (miscellaneous)ArticleClinical trial2712 Endocrinology Diabetes and Metabolism10036 Medical Clinic2724 Internal MedicineCohortmedicineGlycogen storage diseasebusiness
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Correction: Daunorubicin reduces MBNL1 sequestration caused by CUG-repeat expansion and rescues cardiac dysfunctions in a Drosophila model of myotoni…

2018

ABSTRACT Myotonic dystrophy (DM) is a dominantly inherited neuromuscular disorder caused by expression of mutant myotonin-protein kinase (DMPK) transcripts containing expanded CUG repeats. Pathogenic DMPK RNA sequesters the muscleblind-like (MBNL) proteins, causing alterations in metabolism of various RNAs. Cardiac dysfunction represents the second most common cause of death in DM type 1 (DM1) patients. However, the contribution of MBNL sequestration in DM1 cardiac dysfunction is unclear. We overexpressed Muscleblind (Mbl), the Drosophila MBNL orthologue, in cardiomyocytes of DM1 model flies and observed a rescue of heart dysfunctions, which are characteristic of these model flies and resem…

congenital hereditary and neonatal diseases and abnormalitiesRNA StabilityNeuroscience (miscellaneous)Medicine (miscellaneous)MuscleblindGeneral Biochemistry Genetics and Molecular BiologyImmunology and Microbiology (miscellaneous)AnimalsDrosophila ProteinsMyotonic DystrophyMyocytes CardiacRNA MessengerDaunorubicinCorrectionNuclear ProteinsReproducibility of ResultsHeartSurvival AnalysisAlternative SplicingDisease Models AnimalDrosophila melanogasterTrinucleotide repeat disorderDrosophilaTrinucleotide Repeat ExpansionResearch ArticleProtein BindingDisease Models & Mechanisms
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Evaluation of the risk factors of asymptomatic vertebral fractures in postmenopausal women with osteopenia at the femoral neck

2016

To identify risk factors of asymptomatic vertebral fracture (aVF) in postmenopausal women with osteopenia at the femoral neck and to evaluate the association between the number of aVFs and the risk of major and hip osteoporotic fracture calculated with the FRAX(®) algorithm.Epidemiological case-series study with data collected transversally.728 postmenopausal women with osteopenia were included: 284 (39.0%) had aVF, of whom 200 (70.4%) had prior fragility fractures (FF). The likelihood of having an osteoporotic fracture in the next 10 years increased significantly with the number of aVF. The percentage of women with height loss, which was assessed as the difference between the greatest heig…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyFRAXBone density030209 endocrinology & metabolismRisk AssessmentAsymptomaticGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineBone DensityRisk FactorsEpidemiologymedicineHumanscardiovascular diseases030212 general & internal medicineAgedRetrospective StudiesFemoral neckFemur NeckHip Fracturesbusiness.industryObstetrics and GynecologyRetrospective cohort studyMiddle Agedmedicine.diseaseSurgeryPostmenopauseOsteopeniaBone Diseases Metabolicmedicine.anatomical_structureSpainMultivariate AnalysisSpinal FracturesFemalemedicine.symptombusinessRisk assessmentAlgorithmsOsteoporotic Fractures
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